Chronic Traumatic Encephalopathy (CTE) is a traumatic injury of the brain that results in progressive degenerative brain disease. The pathology of Chronic Traumatic Encephalopathy (CTE) is known by the buildup of abnormal proteins, also known as “tau.”
It is clinically characterized by the following symptoms:
- Mood swings
- Coordination issues
- Memory loss
Chronic Traumatic Encephalopathy (CTE) is commonly found among athletes involved in sporting activities where concussive head hits are likely to occur, such as boxing, soccer, wrestling, and football. However, other activities such as mining and military duties can also cause these concussive or sub-concussive head impacts. In these occupations, notorious explosives blasts are the problem.
Despite all the ongoing pharmaceutical research to obtain a definitive CTE treatment regime, there is currently no proven CTE treatment regimen. Conventional treatment has been administered to target its symptoms. However, one CTE treatment option is quickly gaining ground as a definitive treatment and cure for CTE: the NAD treatment option.
In 2012, star linebacker Junior Seau committed suicide and died. He was diagnosed on autopsy and found Chronic Traumatic Encephalopathy (CTE) by the National Institute of Health. Since then, this CTE condition has been reported in other suicidal and non-suicidal deaths of many National Football League players.
In addition to the suicidal tendencies suffered by patients with Chronic Traumatic Encephalopathy (CTE), other observable symptoms include:
- Chronic depression
- Aggressive and irrational behavior
- Lack of concentration
- Memory loss or dementia
- Locomotive imbalance and deficiencies
However, these symptoms are not definitive and restricted to Chronic Traumatic Encephalopathy (CTE) because they can also be caused by other clinical conditions such as stress, sleep disorders and emotional triggers like anxiety and fear.
Chronic Traumatic Encephalopathy (CTE) symptoms develop over a long period of time, although biological changes can be seen within a single traumatic brain injury event. Anyone who receives repetitive impacts to the head is at risk for CTE. Most cases of CTE are found in athletes participating in contact sports.
The diagnosis of Chronic Traumatic Encephalopathy (CTE) is still only definitive by autopsy, which makes the development of a curative CTE treatment difficult. In post-mortem diagnosis, dementia caused by CTE is notorious for having tangles of abnormal protein tau in the brain.
Furthermore, medical Chronic Traumatic Encephalopathy (CTE) is also nearly spotted definitively thorough medical history, mental status testing, neurological examinations, and brain imaging—majorly to rule out other possible causes of the aforementioned symptoms. Yet, individuals originally suspected of having Chronic Traumatic Encephalopathy (CTE) in life have tested negative for Chronic Traumatic Encephalopathy (CTE) during post-mortem examinations.
Chronic Traumatic Encephalopathy (CTE) does not have definitive curative treatment measure at present. However, proper management of the symptoms proves effective with far-reaching beneficial effects on victims. Prevention of repeated concussive, sub-concussive and non-concussive head impacts remains the best measure in managing Chronic Traumatic Encephalopathy (CTE). A supportive environment also helps manage the spontaneity in these patients’ behaviors.
Nicotinamide Adenine Dinucleotide (NAD/NADH) Therapy
The Nicotinamide Adenine Dinucleotide (NAD/NADH) therapy is one of the most promising treatments for CTE treatment. This enzyme has been shown to promote neurogenesis even after trauma. It does this by inducing new positive connections in the brain while neutralizing some brain degenerative substances.
The NAD Therapy for CTE treatment is a novel addition to the treatment options. An uncommon therapeutic market commodity, it has not undergone major clinical trials for its long-term effect. NAD, or Nicotinamide Adenine Dinucleotide, is a natural co-enzyme of niacin involved in body energy generation through a complex biological process.
Through these processes, Nicotinamide Adenine Dinucleotide (NAD) catalyzes the extraction of cellular energy from the food we ingest. Despite its importance in generating cellular energy that is necessary for normal brain function, it can be depleted by lifestyle habits such as illicit drug abuse, post-traumatic stress, depression, fear, anxiety, Alzheimer, Parkinson disease, neurodegenerative diseases, aging and Chronic Traumatic Encephalopathy (CTE).
NAD Therapy requires the use of a slow intravenous transmission of the co-enzyme into the bloodstream. This allows the substance to bypass the stomach (where analgesic medication breaks down) and travel directly to the brain. This provides the individual with a boost of energy for enhanced mood and awareness. Keep in mind that the energy derives from natural sources, not other substances. There are also claims of its effects on slowing the aging process.
The Nicotinamide Adenine Dinucleotide (NAD/NADH) therapy is available, and the resources required for both carrying it out and maintaining the results can be found at Springfield Addiction Clinic. A mouse click on this hyperlink will lead you to abundant resources and a safe Nicotinamide Adenine Dinucleotide (NAD/NADH) therapy for curing degenerative brain diseases, addiction challenges and Chronic Traumatic Encephalopathy (CTE).
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